Download Spinal Angiomas: Advances in Diagnosis and Therapy by Guy Lazorthes (auth.), Professor Dr. Dr. h.c. Hans Werner PDF

By Guy Lazorthes (auth.), Professor Dr. Dr. h.c. Hans Werner Pia, René Djindjian (eds.)

The speedy improvement of diagnostic and healing systems within the administration of spinal angiomas has unfolded new chances and supplied greater percentages for the sufferers involved. the best impetus to this professional­ gress used to be given by means of the advent of selective and superselective spinal angiography, microsurgical method, and embolization. those refined ideas and the ability required for his or her use are faraway from being regimen within the neurosurgical and neuroradiologic departments. inspite of the rarity of spinal angiomas, the applying of the above-mentioned tactics is the prerequisite for making improvements to early prognosis and giving well timed enough deal with­ ment. hold up in analysis and therapy are nonetheless the most reason for unsatisfac­ tory effects. within the final 10 -20 years, a number of teams in Europe and the us have performed vital and basic paintings in introducing and constructing the diganos­ tic and healing armamentarium. according to the pioneering paintings in their academics and the vintage contribution of Wyburn - Mason in 1943, they concurrently better the morphologic, physiologic, and scientific foundation of our wisdom. even though development is happening and plenty of difficulties need to be solved, the overall ideas of medical prognosis, operative therapy, and embolization were laid down and are to be released in a unique monograph in this topic.

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Extra resources for Spinal Angiomas: Advances in Diagnosis and Therapy

Sample text

X 5) blastoma of the cauda with intradural AVM and necrotizing myelopathy of the thoracic cord combined with cervical syringomyelia was seen in a male aged 25 years (Fig. 9). Although it has been argued that the syringomyelic cavity might be due to transudation of plasma from the tumor vessels, the separate occurrence of syringomyelia and AVM supports the view that they constitute separate congenital anomalies (Rubinstein, 1972; Wyburn-Mason, 1943), representing parts of dysgenetic syndromes (phacomatoses) that are closely allied to each other.

X 260, (C) Extravasation of fibrinoid and hyalin masses in necrotic grey matter of thoracic cord. G. x 100, (D) Fibrinoid necrosis and mineralization of teleangiectatic vessels in necrotic gray matter. V. , 1970), followed by progressive radiculospinal symptoms. , 1969). There is a striking increase and proliferation of intramedullary blood vessels within and outside the necrotic areas (Fig. 8). These abundant vascular channels, the histology of which often does not allow their designation as veins or arteries, show fibrinoid necrosis (Fig.

7A), often associated with pial vascular anomalies. The lesions of the radicular tributaries are often associated with severe damage to the lumbosacral nerve roots ranging from flaky or segmental demyelination to complete nerve fibre depletion and fibrous scarring (Figs. 4C, D, 9C, D). These radicular changes apparently occur before damage to the cord and thus may explain the initial clinical signs and symptoms. , 1970) and "dysbasia intermittens spinalis" 35 Fig . 7. "Angiodysgenetic" myelopathies.

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