By P. O. Madsen, P. Drescher, T. C. Gasser (auth.), Prof. Dr. Wolfgang Weidner, Prof. Dr. Paul O. Madsen, Prof. Dr. Hans Gerd Schiefer (eds.)
Prostatitis remains to be an incredible medical enigma. during this ebook, all glossy features of etiology, pathogenesis, analysis and treatment are seriously evaluated. Of precise curiosity to the readers could be the sleek diagnostic administration of power prostatitis and the controversial healing method of power nonbacterial prostatitis. From the contents: Radical Transurethral Prostatectomy for continual Bacterial Prostatitis; Etiology, Pathogenesis, and Inflammatory Reactions in persistent Bacterial Prostatitis; remedy of Nonbacterial Prostatitis; Ultrasonographic positive factors of Prostatitis.
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Additional info for Prostatitis: Etiopathology, Diagnosis and Therapy
The central zone is closely associated with other W olffian-derived structures such as the seminal vesicles and distal vas deferens, and it is noteworthy that the central zone encloses the lower part of the vesicles and the ejaculatory ducts. This part of the gland is developmentally homologous to the paraurethral glands of the female, and the acinar epithelium of the central zone has fewer androgen receptors and appears less androgen dependent than the epithelium of the peripheral zone; the acini develop from the otherwise undifferentiated fibromuscular stroma before puberty unlike those of the peripheral zone, which are much more androgen dependent and await the stimulus of puberty before development.
1988; Virkola et al. 1988). , Enterococcus spp, are less clear, and several mechanisms of adhesion have been detected. Grampositive bacteria bind to N -acetylneuraminic acids of glycoproteins, to galactosamine or fJ-galactoside residues, or to fibronectin-rich cells, using their teichoic or lipoteichoic acids. An adhesin called aggregation substance, which enables cell-cell contact between bacteria, mediates adhesion of enterococci to eukaryotic cells. The adhesin is of proteinaceous nature, is located on the surface of the bacteria, and can be seen as hairlike structures using an electron microscope (Andriole 1987; Hasty et al.
This exopolysaccharide film can protect the bacteria from the effects of antibiotic and the host defence mechanisms, so permitting their persistence within the gland (1990). Whilst loculation of organisms within the gland due to duct obstruction by oedema or fibrosis occurs in chronic prostatitis, this glycocalix formation can also explain the phenomenon of chronic prostatitis with recrudescence at intervals. The glycocalixenclosed bacteria are closely adherent to the acinar and duct epithelium and therefore difficult to dislodge by prostatic massage.
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