By Roy O. Greep
Contemporary growth in Hormone examine, quantity 30 provides the court cases of the 1973 Laurentian Hormone convention held at Mont Tremblant, Province of Quebec, Canada on August 26-31, 1973. The booklet offers papers at the law of the gonadotropins in primates, their neutralization by means of particular antibodies, and their function in oocyte maturation. The textual content additionally contains papers at the enzymatic interconversion of estrogens and at the suggestion of the mechanism of steroid hormone motion. Papers at the thermogenic motion of thyroid hormone regarding energetic sodium delivery also are thought of. The ebook additional tackles papers at the constitution, heterogeneity, and job of a few of the lesser identified hormones or hormone-like elements, similar to the somatomedins A, B, and C, the epidermal development issue, the insect hormones, parathyroid hormone, 1,25-dihydroxycholecalciferol, and prostaglandins. Endocrinologists and physiologists will locate the textual content necessary.
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Additional info for Proceedings of the 1973 Laurentian Hormone Conference
Despite the fact that this and most other animals in this series lactated for a time after the operation, no notable, chronic changes in circulating prolactin levels were observed. The most salient conclusion to be drawn from these very recent studies is that the sites of both the positive and negative feedback actions of estrogen and, hence, of the central components of the control systems which govern tonic and surge secretion of the gonadotropic hormones must be resident within the medial basal hypothalamicohypophysial unit.
A. McCracken, J. Reprod. Fert. (1973) 34, 357. J. W. McArthur: To extend Dr. Knobil's observations to another species, I can report that the release of L H is induced in human volunteers given either diethylstilbestrol (DES) by mouth or Premarin by vein during the follicular phase of the cycle. The administration of either 20 mg of D E S or 40 mg of Premarin resulted in a premature release of L H , a delay in the occurrence of the spontaneous L H peak, and prolongation of the cycle. If the same preparations were given during the luteal phase, none of these phenomena occurred [J.
Chobsieng, U. Zor, M. Fridkin, and H. R. Lindner, Biochem. Biophys. Res. Commun. (1973) 55, 623]. Perhaps such an antiserum could be used to examine whether in the monkey estradiol exerts its possible feedback directly on the pituitary or via the hypothalamus. We completely agree with Dr. McCracken's work on the effect of prostaglandin. We found that prostaglandin E 2 had no effect on L H release from rat hemipituitaries incubated in vitro, b u t did stimulate L H secretion in vivo, presumably by an effect on the hypothalamus.
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