Download Parasite Antigens in Protection, Diagnosis and Escape by R. J. Terry (auth.), R. M. E. Parkhouse MD (eds.) PDF

By R. J. Terry (auth.), R. M. E. Parkhouse MD (eds.)

An anticipated 2-3 billion humans within the much less constructed international locations be afflicted by infections, frequently a number of, attributable to a number of parasitic organisms. those infections are usually debilitat­ ing instead of deadly, and the toll in human distress is fearsome. To this can be additional the superiority of comparable illnesses in do­ mestic animals, which lower offers of animal professional tein. because the global inhabitants raises, the already huge, immense challenge additionally maintains to develop. The assets of the fewer constructed countries are insufficient for fixing the matter, and within the de­ veloped international locations an absence of curiosity in tropical ailments has intended low precedence for study. contemporary methodological advances now increase the genuine probability of a scientific and effec­ tive assault upon those ailments - hybridoma and recombinant nucleic acid applied sciences. the mix ofthese with the nonetheless invaluable medical, parasitological and imrnunological in­ formation allows a logical, deliberate and reasonable method of analysis and therapy. The imperative target ofthese modem tech­ niques is to outline antigens with reference to prognosis, security and pathology. in terms of a few ailments, paintings has already started alongside those strains; in relation to others, wisdom lags some distance at the back of. This quantity represents a precis of present wisdom a couple of broad, consultant spectrum of tropical illnesses. there's substantial universal flooring among the several infections as regards goals and the tools for attaining them.

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Example text

Polymorphonucleocytes were shown to be able to kill non-pathogenic E. histolytica trophozoites (GUERRANT et al. 1981). Amoebae of a low-virulence strain which were surrounded by PMNs (more than 300/amoeba) were fragmented and ingested by the PMN s by means that appeared to be independent of oxygen or of complement. In contrast, contact with more virulent amoebae (defined by their ability to cause hepatic abscesses in hamsters and destroy cell monolayers in tissue culture) caused lass ofPMN motility, PMN degranulation, death and occasional phagocytosis by the amoebae.

Histolytica trophozoite antigen. Although cross-reactivity between strains of E. histolytica is relied upon, it has been shown (CHANG et al. 1979; ISHAQ and PADMA 1980) that different isolates have distinct, as weB as shared, antigens. Cross-re action with other Entamoeba strains and with related parasites does not seem, empiricaBy, to be a big problem. Low cross-reactivity has been reported with Entamoeba moshkovskii, Entamoeba coli, the Laredo strain and Giardia lamblia (GOLDMAN et al. 1962; PARKHOUSE et al.

Amoebae of a low-virulence strain which were surrounded by PMNs (more than 300/amoeba) were fragmented and ingested by the PMN s by means that appeared to be independent of oxygen or of complement. In contrast, contact with more virulent amoebae (defined by their ability to cause hepatic abscesses in hamsters and destroy cell monolayers in tissue culture) caused lass ofPMN motility, PMN degranulation, death and occasional phagocytosis by the amoebae. PMNs which were in the vicinity ofvirulent amoebae, but not in contact with them, remained alive.

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