By J. Larry Jameson, Joseph Loscalzo
The authority of Harrison's in a convenient, full-color paperback committed solely to Nephrology and Acid-Base problems A Doody's center name for 2011! four superstar DOODY'S evaluate! "In the Harrison's culture, this e-book integrates pathophysiology with scientific administration whereas outlining discoveries in genetics and molecular biology. It contains a few vital chapters that aren't present in related books: one on variation of the kidney to renal harm (including elements and mechanisms of renal affliction development) and one on hyperuricemia and gout. The introductory bankruptcy at the easy biology of the kidney, masking renal improvement, functionality, and body structure, is particularly good written. This booklet meets the anticipated Harrison's criteria and studying it's a pleasing experience."--Doody's evaluate provider that includes the chapters on Nephrology and Acid-Base issues that seem within the landmark Harrison's ideas of inner drugs, 17e, this compact medical spouse promises the entire most recent wisdom within the box, subsidized through the medical rigor and reliability that experience outlined Harrison's. you will discover assurance that displays the services of well known editors and participants -- provided in a carry-anywhere layout that makes it excellent for the study room, the wards, or the purpose of care. With its ease of use, integral diagnosis-speeding guidance, and acclaimed state of the art thoughts, Harrison's Nephrology and Acid Base issues is a needs to for college kids and clinicians alike. Harrison's Nephrology and Acid-Base issues offers: present, whole assurance of all significant renal-related displays and illnesses, in addition to acid-base and electrolyte problems sixty three high-yield self-assessment Q&A for examination and board overview drawn from Harrison's rules of inner medication Self-Assessment and Board overview, 17e A well timed concentrate on the effect of clinical advances, with a glance at how genetics and molecular biology are remodeling the sector New diagnostic and remedy techniques according to proof from fresh scientific experiences Integration of pathophysiology with medical administration issues in all the disease-oriented chapters precious content material in print shape from the acclaimed Harrison's DVD useful appendix that highlights laboratory values of scientific significance The Harrison’s uniqueness sequence is written through a similar world-renowned writer crew who introduced you Harrison’s rules of inner medication, 17e: Anthony S. Fauci, MD; Eugene Braunwald, MD; Dennis L. Kasper, MD; Stephen L. Hauser, MD; Dan L. Longo, MD; J. Larry Jameson, MD, PhD; Joseph Loscalzo, MD, PhD
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Extra info for Harrison's Nephrology and Acid-Base Disorders
A reduced GFR leads to retention of nitrogenous waste products (azotemia) such as urea and creatinine. 5 g protein/24 h Hydronephrosis Normal size kidneys Intact parenchyma Urologic evaluation Relieve obstruction Bacteria WBC, casts eosinophils Alterations of Renal Function and Electrolytes Normal urinalysis FeNa<1% U osmolality > 500 mosmol Prerenal Azotemia Volume contraction, cardiac failure, vasodilatation, drugs, sepsis, renal vasoconstriction, impaired autoregulation Urine electrolytes Interstitial nephritis Abnormal urinalysis Red blood cells Chronic Renal Failure Symptomatic treatment delay progression If end-stage, prepare for dialysis Pyelonephritis Muddy brown casts, amorphous sediment + protein FeNa>1% U osmolality < 350 mosmol Acute Tubular Necrosis RBC casts Proteinuria Renal artery or vein occlusion Angiogram Renal biopsy Glomerulonephritis or vasculitis Immune complex, anti-GBM disease FIGURE 3-1 Approach to the patient with azotemia.
41 Key diagnostic features of selected diseases in renal biopsy and urinalysis are illustrated, with light, immunoﬂuorescence, A and electron microscopic images. Common urinalysis ﬁndings are also documented. B FIGURE 4-1 Minimal change disease. In minimal change disease, light microscopy is unremarkable (A), while electron microscopy reveals podocyte injury evidenced by complete foot process effacement (B). ) FIGURE 4-2 Focal segmental glomerulosclerosis.
An extensive list of potential drugs and toxins implicated in ATN can be found in Chap. 10. Processes that involve the tubules and interstitium can lead to acute renal failure. , sarcoid, lymphoma, or leukemia). A list of drugs associated with allergic interstitial nephritis can be found in Chap. 17. The urinalysis usually shows mild to moderate proteinuria, hematuria, and pyuria (∼75% of cases) and occasionally white blood cell casts. The ﬁnding of RBC casts in interstitial nephritis has been reported but should prompt a search for glomerular diseases (Fig.
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