By Brian R. Jewell (auth.), Jan Baan Ph.D., Alexander C. Arntzenius M.D., Edward L. Yellin Ph.D. (eds.)
Cardiac Dynamics is the identify of a comparatively younger box of analysis, born from the fruitful interplay among branches of 2 various disciplines: medication and physics. "Dynamics" is the department of physics which offers with the motion of forces on our bodies or debris in movement or at leisure. "Cardiac" pertains to the scientific box of cardiology but additionally to cardiophysiology, either one of that are really good branches of drugs. Narrower than the good tested box of Hemodynamics, Cardiac Dynamics is specific to dynamic phenomena happening in and round the middle. The mathematical remedy of such phenomena, even if, is enormously extra advanced end result of the complex nature of the mechanisms enthusiastic about the cardiac motion. therefore, while hemodynamics is worried with predominantly passive (visco-) elastic constructions - vessels - containing time-variant movement of viscous flui- blood -, the mechanical learn of the guts calls for extra con siderations resembling: energetic elastic elements representing the contractile mechanism of cardiac muscle, complicated geometry and fiber constitution within the myocardial wall, autoregulatory mechanisms, and complex circulate styles linked to valve movement. considered during this mild it isn't spectacular that makes an attempt to explain ventricular pump functionality and to quantify contractile functionality haven't reached the extent of class that is universal in e. g. arterial hemodynamics. for a similar cause, a few of the usually simplified techniques to explain ventricular mechanics did not withstand extra rigorous theoretical, experimental or scientific testing.
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Additional resources for Cardiac Dynamics
They are also in keeping with the results of Huntsman and Joseph (this volume), who found that the force-velocity relation of the central region of cardiac muscle preparations is constant rapidly after the onset of contraction. The influence of different external calcium concentrations on the shape of the sarcomere length-tension relation supports the hypothesis that activation of cardiac muscle is length-dependent (1). It is unlikely that passive restoring forces playa major role in determining the shape of the lengthtension relation as their effect should be calcium-dependent in order to explain the observed change of shape with different calcium concentrations.
Boom HBK, Denier van der Gon 11, Nieuwenhuijs JHM, Schiereck P: Cardiac contractility: actin-myosin interaction as measured from the left ventricular pressure curve. Eur J Cardia I 1:217-224, 1973. 5. Julian FJ: Activation in a skeletal muscle contraction model with a modification for insect fibrilar muscle. Biophys J 9:547-570, 1969. 6. Fletcher R, Powell MJD: A rapid descent method for minimization. Computer J 6(2):163168, 1963. 7. Grood ES, Mates RE: Influence of crossbridge compliance on the force-velocity relation of muscle.
The model fitting procedure did not result either in quantification of the separated preload and contractility parameters. The fit of model and experimentally measured data was quite good (Figure 3). The parameters g, T and D [1,2] could be quantified well but E tended to become infinite, thereby decreasing the amplitude of f(t) to zero. The time course remained identical for all pressure data sets, independent of preload or [Ca + +]. Preload and contractility changes only resulted in changes in the amplitude of the term Qf(t).
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